Figure 3From: Expression of human A53T alpha-synuclein in the rat substantia nigra using a novel AAV1/2 vector produces a rapidly evolving pathology with protein aggregation, dystrophic neurite architecture and nigrostriatal degeneration with potential to model the pathology of Parkinson's diseaseExpression of transgenes in the striatum. Both GFP and human alpha-synuclein were widely expressed throughout the striatum 3 weeks following delivery of AAV1/2 to the substantia nigra. Scale bar in panel A is 500 μm.Back to article page