Fig. 3From: Prion protein oligomers cause neuronal cytoskeletal damage in rapidly progressive Alzheimer’s diseaseExpression of G2L2 and associated proteins in the frontal cortex of experimental cohorts. A & B) Immunoblots showing the expression levels of G2L2, EB-1, α-tubulin, and β-actin. GAPDH served as loading control. C-F) Densitometric quantification for expression of G2L2, EB-1, α-tubulin, and β-actin in spAD (n = 7), rpAD (n = 7), and control (n = 6) cases, as assessed in three technical replicates. No significant differences were found for G2L2 and its associated proteins. Statistical significance was calculated with one-way ANOVA followed by Tukey’s post-hoc testBack to article page