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Fig. 3 | Molecular Neurodegeneration

Fig. 3

From: Prion protein oligomers cause neuronal cytoskeletal damage in rapidly progressive Alzheimer’s disease

Fig. 3

Expression of G2L2 and associated proteins in the frontal cortex of experimental cohorts. A & B) Immunoblots showing the expression levels of G2L2, EB-1, α-tubulin, and β-actin. GAPDH served as loading control. C-F) Densitometric quantification for expression of G2L2, EB-1, α-tubulin, and β-actin in spAD (n = 7), rpAD (n = 7), and control (n = 6) cases, as assessed in three technical replicates. No significant differences were found for G2L2 and its associated proteins. Statistical significance was calculated with one-way ANOVA followed by Tukey’s post-hoc test

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Molecular Neurodegeneration

ISSN: 1750-1326