Human iPSC-derived astrocytes transplanted into the mouse brain undergo morphological changes in response to amyloid-β plaques

Table 2 Details of Human Cases.

Case number	Age	Gender	Aβ phase	Braak stage	PMI	Neuropathological Diagnosis	Type of dementia
1	82	M	5	3	72	AD	AD
2	81	F	5	5	48	AD, CAA, I	AD
3	85	M	5	3	48	AD, CAA, MI	AD-VaD
4	83	M	5	5	24	AD, CAA, I, B	AD-VaD
5	83	F	4	4	24	p-preAD, AGD, CM	0
6	85	F	4	3	24	p-preAD	0
7	87	M	4	3	96	p-preAD, CAA	0
8	72	M	2	3	72	p-preAD, I	0
9	75	M	1	2	48	p-pre-AD, AGD	0
10	66	F	0	0	48	non-AD control, AGD	0
11	62	M	0	0	48	non-AD control	0
12	64	M	0	0	24	non-AD control	0

The table shows the human subjects studied for histology of astrocytes. Indicated are: the age in years, the gender, the Aβ-MTL phase representing the distribution of Aβ deposits in the subfields of the MTL [43], the stage of neurofibrillary tangle pathology according to Braak and Braak [44] (NFT stage), PMI, neuropathological diagnosis and type of dementia. F female, M male, AD Alzheimer’s disease, AD-VaD Alzheimer’s disease plus signs of vascular dementia, p-preAD preclinical AD, non-AD non-demented control, AGD argyrophilic grain disease, B bleeding, CAA cerebrovascular angiopathy, CM carcinoma metastasis, I infarction, MI microinfraction, PMI post-mortem interval

ISSN: 1750-1326