Skip to content



Page 5 of 19

  1. Content type: Research article

    Most sequencing studies in Parkinson’s disease (PD) have focused on either a particular gene, primarily in familial and early onset PD samples, or on screening single variants in sporadic PD cases. To date, th...

    Authors: Bruno A. Benitez, Albert A. Davis, Sheng Chih Jin, Laura Ibanez, Sara Ortega-Cubero, Pau Pastor, Jiyoon Choi, Breanna Cooper, Joel S. Perlmutter and Carlos Cruchaga

    Citation: Molecular Neurodegeneration 2016 11:29

    Published on:

  2. Content type: Research article

    α-Synuclein (α-Syn), a pathological hallmark of Parkinson’s disease (PD), has been recognized to induce the production of interleukin-1β in a process that depends, at least in vitro, on nod-like receptor prote...

    Authors: Yan Zhou, Ming Lu, Ren-Hong Du, Chen Qiao, Chun-Yi Jiang, Ke-Zhong Zhang, Jian-Hua Ding and Gang Hu

    Citation: Molecular Neurodegeneration 2016 11:28

    Published on:

  3. Content type: Research article

    Huntington’s disease (HD) is an incurable hereditary neurodegenerative disorder, which manifests itself as a loss of GABAergic medium spiny (GABA MS) neurons in the striatum and caused by an expansion of the C...

    Authors: Evgeny D. Nekrasov, Vladimir A. Vigont, Sergey A. Klyushnikov, Olga S. Lebedeva, Ekaterina M. Vassina, Alexandra N. Bogomazova, Ilya V. Chestkov, Tatiana A. Semashko, Elena Kiseleva, Lyubov A. Suldina, Pavel A. Bobrovsky, Olga A. Zimina, Maria A. Ryazantseva, Anton Yu. Skopin, Sergey N. Illarioshkin, Elena V. Kaznacheyeva…

    Citation: Molecular Neurodegeneration 2016 11:27

    Published on:

  4. Content type: Research article

    Glaucoma is a complex, multifactorial disease characterised by the loss of retinal ganglion cells and their axons leading to a decrease in visual function. The earliest events that damage retinal ganglion cell...

    Authors: Pete A. Williams, James R. Tribble, Keating W. Pepper, Stephen D. Cross, B Paul Morgan, James E. Morgan, Simon W. M. John and Gareth R. Howell

    Citation: Molecular Neurodegeneration 2016 11:26

    Published on:

  5. Content type: Research article

    Parkinson’s disease (PD) is a progressive neurodegenerative disorder characterized by motor and non-motor symptoms. The cause of the motor symptoms is the loss of dopaminergic neurons in the substantia nigra with...

    Authors: Milena Pinto, Nadee Nissanka, Susana Peralta, Roberta Brambilla, Francisca Diaz and Carlos T. Moraes

    Citation: Molecular Neurodegeneration 2016 11:25

    Published on:

  6. Content type: Research article

    C1q represents the initiating protein of the classical complement cascade, however recent findings indicate pathway independent roles such as developmental pruning of retinal ganglion cell (RGC) axons. Further...

    Authors: Sean M. Silverman, Byung-Jin Kim, Garreth R. Howell, Joselyn Miller, Simon W. M. John, Robert J. Wordinger and Abbot F. Clark

    Citation: Molecular Neurodegeneration 2016 11:24

    Published on:

  7. Content type: Research article

    Interleukin-1 beta (IL-1β) and its key regulator, the inflammasome, are suspected to play a role in the neuroinflammation observed in Alzheimer’s disease (AD); no conclusive data are nevertheless available in ...

    Authors: Marina Saresella, Francesca La Rosa, Federica Piancone, Martina Zoppis, Ivana Marventano, Elena Calabrese, Veronica Rainone, Raffaello Nemni, Roberta Mancuso and Mario Clerici

    Citation: Molecular Neurodegeneration 2016 11:23

    Published on:

  8. Content type: Research article

    Increasing evidence suggests a potential therapeutic benefit of vitamin D supplementation against Alzheimer’s disease (AD). Although studies have shown improvements in cognitive performance and decreases in ma...

    Authors: Véréna Landel, Pascal Millet, Kévin Baranger, Béatrice Loriod and François Féron

    Citation: Molecular Neurodegeneration 2016 11:22

    Published on:

  9. Content type: Research article

    In frontotemporal dementia (FTD) there is a critical lack in the understanding of biological and molecular mechanisms involved in disease pathogenesis. The heterogeneous genetic features associated with FTD su...

    Authors: Raffaele Ferrari, Paola Forabosco, Jana Vandrovcova, Juan A. Botía, Sebastian Guelfi, Jason D. Warren, Parastoo Momeni, Michael E. Weale, Mina Ryten and John Hardy

    Citation: Molecular Neurodegeneration 2016 11:21

    Published on:

  10. Content type: Erratum

    Authors: Wendy E. Heywood, Daniela Galimberti, Emily Bliss, Ernestas Sirka, Ross W. Paterson, Nadia K. Magdalinou, Miryam Carecchio, Emma Reid, Amanda Heslegrave, Chiara Fenoglio, Elio Scarpini, Jonathan M. Schott, Nick C. Fox, John Hardy, Kailash P. Bhatia, Simon Heales…

    Citation: Molecular Neurodegeneration 2016 11:20

    Published on:

    The original article was published in Molecular Neurodegeneration 2015 10:64

  11. Content type: Research article

    The metalloprotease meprin β cleaves the Alzheimer’s Disease (AD) relevant amyloid precursor protein (APP) as a β-secretase reminiscent of BACE-1, however, predominantly generating N-terminally truncated Aβ2-x...

    Authors: Caroline Schönherr, Jessica Bien, Simone Isbert, Rielana Wichert, Johannes Prox, Hermann Altmeppen, Sathish Kumar, Jochen Walter, Stefan F. Lichtenthaler, Sascha Weggen, Markus Glatzel, Christoph Becker-Pauly and Claus U. Pietrzik

    Citation: Molecular Neurodegeneration 2016 11:19

    Published on:

  12. Content type: Research article

    Alzheimer disease (AD) and other tauopathies develop cerebral intracellular inclusions of hyperphosphorylated tau. Epidemiological and experimental evidence suggests a clear link between type 2 diabetes mellit...

    Authors: Erica Barini, Odetta Antico, Yingjun Zhao, Francesco Asta, Valter Tucci, Tiziano Catelani, Roberto Marotta, Huaxi Xu and Laura Gasparini

    Citation: Molecular Neurodegeneration 2016 11:16

    Published on:

  13. Content type: Methodology

    While many biological studies can be performed on cell-based systems, the investigation of molecular pathways related to complex human dysfunctions – e.g. neurodegenerative diseases – often requires long-term ...

    Authors: Matteo Cornaglia, Gopalan Krishnamani, Laurent Mouchiroud, Vincenzo Sorrentino, Thomas Lehnert, Johan Auwerx and Martin A. M. Gijs

    Citation: Molecular Neurodegeneration 2016 11:17

    Published on:

  14. Content type: Research article

    Prions and amyloid-β (Aβ) oligomers trigger neurodegeneration by hijacking a poorly understood cellular signal mediated by the prion protein (PrP) at the plasma membrane. In early zebrafish embryos, PrP-1-depe...

    Authors: Emily Sempou, Emiliano Biasini, Alejandro Pinzón-Olejua, David A. Harris and Edward Málaga-Trillo

    Citation: Molecular Neurodegeneration 2016 11:18

    Published on:

  15. Content type: Research article

    A unique feature of the pathological change after spinal cord injury (SCI) is the progressive enlargement of lesion area, which usually results in cavity formation and is accompanied by reactive astrogliosis a...

    Authors: Hong Fan, Kun Zhang, Lequn Shan, Fang Kuang, Kun Chen, Keqing Zhu, Heng Ma, Gong Ju and Ya-Zhou Wang

    Citation: Molecular Neurodegeneration 2016 11:14

    Published on:

  16. Content type: Research article

    Parkinson’s disease (PD) is a motor disease associated with the degeneration of dopaminergic neurons of the substantia nigra pars compacta. p53 is a major neuronal pro-apoptotic factor that is at the center of gr...

    Authors: Eric Duplan, Cécile Giordano, Frédéric Checler and Cristine Alves da Costa

    Citation: Molecular Neurodegeneration 2016 11:13

    Published on:

  17. Content type: Methodology

    Deep brain stimulation (DBS) of the subthalamic nucleus (STN) or the internal segment of the globus pallidus (GPi) has been established as a highly effective symptomatic therapy for Parkinson’s disease (PD). A...

    Authors: Affif Zaccaria, Ali Bouamrani, Stephan Chabardès, Michèle El Atifi, Eric Seigneuret, Johannes A. Lobrinus, Michel Dubois-Dauphin, François Berger and Pierre R. Burkhard

    Citation: Molecular Neurodegeneration 2016 11:12

    Published on:

  18. Content type: Research article

    Prion diseases are characterized by the accumulation in the central nervous system of an abnormally folded isoform of the prion protein, named PrPSc. Aggregation of PrPSc into oligomers and fibrils is critically ...

    Authors: Thibaut Imberdis, Adeline Ayrolles-Torro, Alysson Duarte Rodrigues, Joan Torrent, Maria Teresa Alvarez-Martinez, Gabor G. Kovacs, Jean-Michel Verdier, Mike Robitzer and Véronique Perrier

    Citation: Molecular Neurodegeneration 2016 11:11

    Published on:

  19. Content type: Research article

    A disintegrin and metalloproteinase with thrombospondin motifs (ADAMTS) proteoglycanases are specialized in the degradation of chondroitin sulfate proteoglycans and participate in mechanisms mediating neuropla...

    Authors: Sighild Lemarchant, Yuriy Pomeshchik, Iurii Kidin, Virve Kärkkäinen, Piia Valonen, Sarka Lehtonen, Gundars Goldsteins, Tarja Malm, Katja Kanninen and Jari Koistinaho

    Citation: Molecular Neurodegeneration 2016 11:10

    Published on:

    The Erratum to this article has been published in Molecular Neurodegeneration 2016 11:15

  20. Content type: Research article

    Mutations in the fused in sarcoma (FUS) gene have been linked to amyotrophic lateral sclerosis (ALS). ALS patients with FUS mutations exhibit neuronal cytoplasmic mislocalization of the mutant FUS protein. ALS pa...

    Authors: Su Min Lim, Won Jun Choi, Ki-Wook Oh, Yuanchao Xue, Ji Young Choi, Sung Hoon Kim, Minyeop Nahm, Young-Eun Kim, Jinhyuk Lee, Min-Young Noh, Seungbok Lee, Sejin Hwang, Chang-Seok Ki, Xiang-Dong Fu and Seung Hyun Kim

    Citation: Molecular Neurodegeneration 2016 11:8

    Published on:

  21. Content type: Research article

    Substantial evidence suggests that amyloid-β (Aβ) species induce oxidative stress and cerebrovascular (CV) dysfunction in Alzheimer’s disease (AD), potentially contributing to the progressive dementia of this ...

    Authors: Matthew R. Reynolds, Itender Singh, Tej D. Azad, Brandon B. Holmes, Phillip B. Verghese, Hans H. Dietrich, Marc Diamond, Guojun Bu, Byung Hee Han and Gregory J. Zipfel

    Citation: Molecular Neurodegeneration 2016 11:9

    Published on:

  22. Content type: Research article

    Despite decades of intensive research, to date, there is no accepted diagnosis for Parkinson’s disease (PD) based on biochemical analysis of blood or CSF. However, neurodegeneration in the brains of PD patient...

    Authors: Nour K. Majbour, Nishant N. Vaikath, Karin D. van Dijk, Mustafa T. Ardah, Shiji Varghese, Louise B. Vesterager, Liliana P. Montezinho, Stephen Poole, Bared Safieh-Garabedian, Takahiko Tokuda, Charlotte E. Teunissen, Henk W. Berendse, Wilma D. J. van de Berg and Omar M. A. El-Agnaf

    Citation: Molecular Neurodegeneration 2016 11:7

    Published on:

  23. Content type: Research article

    Mitochondrial dysfunction, oxidative stress and their interplay are core pathological features of Parkinson’s disease. In dopaminergic neurons, monoamines and their metabolites provide an additional source of ...

    Authors: Mária Baranyi, Pier Francesca Porceddu, Flóra Gölöncsér, Szabina Kulcsár, Lilla Otrokocsi, Ágnes Kittel, Annalisa Pinna, Lucia Frau, Paul B. Huleatt, Mui-Ling Khoo, Christina L. L. Chai, Petra Dunkel, Peter Mátyus, Micaela Morelli and Beáta Sperlágh

    Citation: Molecular Neurodegeneration 2016 11:6

    Published on:

  24. Content type: Research article

    Lrrk2, a gene linked to Parkinson’s disease, encodes a large scaffolding protein with kinase and GTPase activities implicated in vesicle and cytoskeletal-related processes. At the pres...

    Authors: Elisa Belluzzi, Adriano Gonnelli, Maria-Daniela Cirnaru, Antonella Marte, Nicoletta Plotegher, Isabella Russo, Laura Civiero, Susanna Cogo, Maria Perèz Carrion, Cinzia Franchin, Giorgio Arrigoni, Mariano Beltramini, Luigi Bubacco, Franco Onofri, Giovanni Piccoli and Elisa Greggio

    Citation: Molecular Neurodegeneration 2016 11:1

    Published on:

  25. Content type: Research article

    The receptor for advanced glycation end products (RAGE) has been found to interact with amyloid β (Aβ). Although RAGE does not have any kinase motifs in its cytosolic domain, the interaction between RAGE and A...

    Authors: Yoonhee Kim, Chaeyoung Kim, Sung Min Son, Hyundong Song, Hyun Seok Hong, Sun-ho Han and Inhee Mook-Jung

    Citation: Molecular Neurodegeneration 2016 11:4

    Published on:

  26. Content type: Research article

    Alzheimer’s disease (AD) is the most frequent form of dementia in the elderly and no effective treatment is currently available. The mechanisms triggering AD onset and progression are still imperfectly dissect...

    Authors: Mickael Audrain, Romain Fol, Patrick Dutar, Brigitte Potier, Jean-Marie Billard, Julien Flament, Sandro Alves, Marie-Anne Burlot, Gaelle Dufayet-Chaffaud, Alexis-Pierre Bemelmans, Julien Valette, Philippe Hantraye, Nicole Déglon, Nathalie Cartier and Jérome Braudeau

    Citation: Molecular Neurodegeneration 2016 11:5

    Published on:

  27. Content type: Short report

    The discovery that heterozygous missense mutations in the gene encoding triggering receptor expressed on myeloid cells 2 (TREM2) are risk factors for Alzheimer’s disease (AD), with only the apolipoprotein E (A...

    Authors: Amanda Heslegrave, Wendy Heywood, Ross Paterson, Nadia Magdalinou, Johan Svensson, Per Johansson, Annika Öhrfelt, Kaj Blennow, John Hardy, Jonathan Schott, Kevin Mills and Henrik Zetterberg

    Citation: Molecular Neurodegeneration 2016 11:3

    Published on:

  28. Content type: Methodology

    Apolipoprotein E (ApoE) is a major cholesterol carrier and plays an important role in maintaining lipid homeostasis both in the periphery and brain. Human APOE gene is polymorphic at two single nucleotides (rs429...

    Authors: Li Zhong, Yong-Zhuang Xie, Tian-Tian Cao, Zongqi Wang, Tingting Wang, Xinxiu Li, Rui-Chi Shen, Huaxi Xu, Guojun Bu and Xiao-Fen Chen

    Citation: Molecular Neurodegeneration 2016 11:2

    Published on:

  29. Content type: Review

    In Alzheimer Disease (AD), the mechanistic connection of the two major pathological hallmarks, namely deposition of Amyloid-beta (Aβ) in the form of extracellular plaques, and the pathological changes of the i...

    Authors: Hans Zempel and Eva-Maria Mandelkow

    Citation: Molecular Neurodegeneration 2015 10:68

    Published on:

  30. Content type: Research article

    Beclin 1 is a key regulator of multiple trafficking pathways, including autophagy and receptor recycling in yeast and microglia. Decreased beclin 1 levels in the CNS result in neurodegeneration, an effect attr...

    Authors: Caitlin E. O’Brien, Liana Bonanno, Hui Zhang and Tony Wyss-Coray

    Citation: Molecular Neurodegeneration 2015 10:69

    Published on:

  31. Content type: Research article

    Although the genetic cause for Huntington’s disease (HD) has been known for over 20 years, the mechanisms that cause the neurotoxicity and behavioral symptoms of this disease are not well understood. One hypot...

    Authors: Matthew B. Veldman, Yesenia Rios-Galdamez, Xiao-Hong Lu, Xiaofeng Gu, Wei Qin, Song Li, X. William Yang and Shuo Lin

    Citation: Molecular Neurodegeneration 2015 10:67

    Published on:

  32. Content type: Research article

    Parkinson’s disease (PD) and amyotrophic lateral sclerosis (ALS) are both neurodegenerative diseases leading to impaired execution of movement. α-Synuclein plays a central role in the pathogenesis of PD wherea...

    Authors: Anika M. Helferich, Wolfgang P. Ruf, Veselin Grozdanov, Axel Freischmidt, Marisa S. Feiler, Lisa Zondler, Albert C. Ludolph, Pamela J. McLean, Jochen H. Weishaupt and Karin M. Danzer

    Citation: Molecular Neurodegeneration 2015 10:66

    Published on:

  33. Content type: Research article

    Presynaptic terminals contribute to cognitive reserve, balancing the effects of age-related pathologies on cognitive function in the elderly. The presynaptic protein Munc18-1, alternatively spliced into long (...

    Authors: Alfredo Ramos-Miguel, Christa Hercher, Clare L. Beasley, Alasdair M. Barr, Thomas A. Bayer, Peter Falkai, Sue E. Leurgans, Julie A. Schneider, David A. Bennett and William G. Honer

    Citation: Molecular Neurodegeneration 2015 10:65

    Published on:

  34. Content type: Research article

    Currently there are no effective treatments for many neurodegenerative diseases. Reliable biomarkers for identifying and stratifying these diseases will be important in the development of future novel therapie...

    Authors: Wendy E. Heywood, Daniela Galimberti, Emily Bliss, Ernestas Sirka, Ross W. Paterson, Nadia K. Magdalinou, Miryam Carecchio, Emma Reid, Amanda Heslegrave, Chiara Fenoglio, Elio Scarpini, Jonathan M. Schott, Nick C. Fox, John Hardy, Kailash Bahtia, Simon Heales…

    Citation: Molecular Neurodegeneration 2015 10:64

    Published on:

    The Erratum to this article has been published in Molecular Neurodegeneration 2016 11:20

  35. Content type: Research article

    Parkinson’s disease (PD) is characterized by progressive loss of midbrain dopaminergic neurons, resulting in motor dysfunctions. While most PD is sporadic in nature, a significant subset can be linked to eithe...

    Authors: Seon-Heui Cha, Yu Ree Choi, Cheol-Ho Heo, Seo-Jun Kang, Eun-Hye Joe, Ilo Jou, Hwan-Myung Kim and Sang Myun Park

    Citation: Molecular Neurodegeneration 2015 10:63

    Published on:

  36. Content type: Research article

    L-methionine, the principal sulfur-containing amino acid in proteins, plays critical roles in cell physiology as an antioxidant and in the breakdown of fats and heavy metals. Previous studies suggesting the us...

    Authors: Cheril Tapia-Rojas, Carolina B. Lindsay, Carla Montecinos-Oliva, Macarena S. Arrazola, Rocio M. Retamales, Daniel Bunout, Sandra Hirsch and Nibaldo C. Inestrosa

    Citation: Molecular Neurodegeneration 2015 10:62

    Published on:

  37. Content type: Research article

    HIV-1-infected and/or immune-activated microglia and macrophages are pivotal in the pathogenesis of HIV-1-associated neurocognitive disorders (HAND). Glutaminase, a metabolic enzyme that facilitates glutamate ...

    Authors: Beiqing Wu, Yunlong Huang, Alexander L. Braun, Zenghan Tong, Runze Zhao, Yuju Li, Fang Liu and Jialin C. Zheng

    Citation: Molecular Neurodegeneration 2015 10:61

    Published on:

  38. Content type: Research article

    Dendritic simplification, a key feature of the neurodegenerative triad of Alzheimer’s disease (AD) in addition to spine changes and neuron loss, occurs in a region-specific manner. However, it is unknown how c...

    Authors: Nataliya Golovyashkina, Lorène Penazzi, Carlo Ballatore, Amos B. Smith III, Lidia Bakota and Roland Brandt

    Citation: Molecular Neurodegeneration 2015 10:60

    Published on:

  39. Content type: Research article

    Ceruloplasmin, a ferroxidase present in cerebrospinal fluid (CSF), plays a role in iron homeostasis protecting tissues from oxidative damage. Its reduced enzymatic activity was reported in Parkinson’s disease ...

    Authors: Marco Barbariga, Flavio Curnis, Annapaola Andolfo, Alan Zanardi, Massimo Lazzaro, Antonio Conti, Giuseppe Magnani, Maria Antonietta Volontè, Laura Ferrari, Giancarlo Comi, Angelo Corti and Massimo Alessio

    Citation: Molecular Neurodegeneration 2015 10:59

    Published on:

  40. Content type: Research article

    Preventing or reducing amyloid-beta (Aβ) accumulation in the brain is an important therapeutic strategy for Alzheimer’s disease (AD). Recent studies showed that the water channel aquaporin-4 (AQP4) mediates so...

    Authors: Zhiqiang Xu, Na Xiao, Yali Chen, Huang Huang, Charles Marshall, Junying Gao, Zhiyou Cai, Ting Wu, Gang Hu and Ming Xiao

    Citation: Molecular Neurodegeneration 2015 10:58

    Published on:

  41. Content type: Research article

    Insulin degrading enzyme (IDE) is a major protease of amyloid beta peptide (Aβ), a prominent toxic protein in Alzheimer’s disease (AD) pathogenesis. Previous studies suggested that statins promote IDE secretio...

    Authors: Sung Min Son, Seokjo Kang, Heesun Choi and Inhee Mook-Jung

    Citation: Molecular Neurodegeneration 2015 10:56

    Published on:

  42. Content type: Research article

    Amyotrophic Lateral Sclerosis is characterized by a focal onset of symptoms followed by a progressive spread of pathology that has been likened to transmission of infectious prions. Cell-to-cell transmission o...

    Authors: Rafaa Zeineddine, Jay F. Pundavela, Lisa Corcoran, Elise M. Stewart, Dzung Do-Ha, Monique Bax, Gilles Guillemin, Kara L. Vine, Danny M. Hatters, Heath Ecroyd, Christopher M. Dobson, Bradley J. Turner, Lezanne Ooi, Mark R. Wilson, Neil R. Cashman and Justin J. Yerbury

    Citation: Molecular Neurodegeneration 2015 10:57

    Published on:

  43. Content type: Research article

    Intracellular accumulation of tau as neurofibrillary tangles (NFTs) is the hallmark of Alzheimer’s disease (AD) as well as in other tauopathies. Tau is present not only in the cytoplasm but also in the extrace...

    Authors: Kaoru Yamada, Tirth K. Patel, Katja Hochgräfe, Thomas E. Mahan, Hong Jiang, Floy R. Stewart, Eva-Maria Mandelkow and David M. Holtzman

    Citation: Molecular Neurodegeneration 2015 10:55

    Published on:

  44. Content type: Commentary

    Affinity-based techniques, both for enrichment or depletion of proteins of interest, suffer from unwanted interactions between the bait or matrix material and molecules different from the original target. This...

    Authors: Ramona Günther, Eberhard Krause, Michael Schümann, Ingolf E. Blasig and Reiner F. Haseloff

    Citation: Molecular Neurodegeneration 2015 10:53

    Published on:

  45. Content type: Review

    In the past five years, a series of large-scale genetic studies have revealed novel risk factors for Alzheimer’s disease (AD). Analyses of these risk factors have focused attention upon the role of immune proc...

    Authors: Manasi Malik, Ishita Parikh, Jared B. Vasquez, Conor Smith, Leon Tai, Guojun Bu, Mary Jo LaDu, David W. Fardo, G. William Rebeck and Steven Estus

    Citation: Molecular Neurodegeneration 2015 10:52

    Published on:

  46. Content type: Research article

    Many neurodegenerative diseases are associated with protein misfolding/aggregation. Treatments mitigating the effects of such common pathological processes, rather than disease-specific symptoms, therefore hav...

    Authors: Xi Chen, Hannah V. McCue, Shi Quan Wong, Sudhanva S. Kashyap, Brian C. Kraemer, Jeff W. Barclay, Robert D. Burgoyne and Alan Morgan

    Citation: Molecular Neurodegeneration 2015 10:51

    Published on:

    The Erratum to this article has been published in Molecular Neurodegeneration 2015 10:54

  47. Content type: Research article

    To identify the causal gene in a multi-incident U.S. kindred with Parkinson’s disease (PD).

    Authors: Ignacio F. Mata, Yongwoo Jang, Chun-Hyung Kim, David S. Hanna, Michael O. Dorschner, Ali Samii, Pinky Agarwal, John W. Roberts, Olga Klepitskaya, David R. Shprecher, Kathryn A. Chung, Stewart A. Factor, Alberto J. Espay, Fredy J. Revilla, Donald S. Higgins, Irene Litvan…

    Citation: Molecular Neurodegeneration 2015 10:50

    Published on:

  48. Content type: Erratum

    Authors: Daniel Sevlever, Fanggeng Zou, Li Ma, Sebastian Carrasquillo, Michael G. Crump, Oliver J. Culley, Talisha A. Hunter, Gina D. Bisceglio, Linda Younkin, Mariet Allen, Minerva M. Carrasquillo, Sigrid B. Sando, Jan O. Aasly, Dennis W. Dickson, Neill R. Graff-Radford, Ronald C. Petersen…

    Citation: Molecular Neurodegeneration 2015 10:49

    Published on:

    The original article was published in Molecular Neurodegeneration 2015 10:18