Fig. 2From: Simple model systems reveal conserved mechanisms of Alzheimer’s disease and related tauopathiesPathways enriched amoung genetic modifiers of AD models. Human homologs of genetic modifiers were analyzed using ShinyGO v0.77 (Ge et al. 2020). Nodes represent the top 30 enriched GO terms. Node size is scaled to the number of genes. Darker nodes are more significantly enriched. Edge thickness is proportional to the number of overlapping genes in each categoryBack to article page